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Bulletin No. 21 - Spring 2005
VirologyPMWS-PCV2-PCVD-PDNS
LOPEZ-SORIA S, SEGALES J, NOFRARIAS M, CALSAMIGLIA M, RAMIREZ H, MINGUEZ A, SARRANO JM, MARIN O, CALLEN A
Genetic influence on the expression of PCV disease [letter].
Veterinary Record, 2004, Volume 155, Nº16, 504
Three paternal genetic lines were used to inseminate sows in two farms (N1 and N2) of a multisite production system. All piglets born of these sows were monitored for mortality from 3 to 21 weeks of age. Post-mortem examination was carried out on each dead piglet, as well as histopathology and in situ hybridization for the detection of PCV2 DNA. Postweaning mortality was 5 and 7.9% in N1 and N2 respectively. About two thirds of mortality were confirmed due to PMWS. The results are summarized in the following table:
| Mortality in farm N1 | Mortality in farm N2 | |
| 100% Pietrain | 1.5% | 2.1% |
| 50% Large White x 50% Pietrain | 4.7% | 5.9% |
| 25% Large White x 75% Duroc | 9.8% | 26.3% |
Significant differences in mortality were observed between the three genetic backgrounds indicating that genetics have a direct effect on postweaning mortality, in this case mostly due to PCV2 infection. However, no direct correlation between PMWS and each breed was established.
