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Bulletin No. 26 - Fall 2006
VirologyPMWS-PCV2-PCVD-PDNS
ROSE N, BLANCHARD P, CARIOLET R, AMENNA N, KERANFLEC'H A, OGER A, JESTIN A, MADEC F
Vaccination parvovirus-rouget chez des truies EOPS infectées expérimentalement par le PCV2: influence sur le développement de la MAP chez la descendance [Influence of parvovirus-erisypelas vaccination of experimentally PCV2-infec Journées Recherche Porcine, 38, 2006, 339-346
Four groups of two SPF sows were included in this study with the following characteristics:
- PCV2-negative non-vaccinated,
- PCV2-positive and vaccinated for parvovirus and erisypelas with two monovalent vaccines (A),
- PCV2-positive and vaccinated for parvovirus and erisypelas with one bivalent vaccine (B),
- PCV2-positive non-vaccinated.
SPF sows were previously inoculated by the oronasal and intrauterine routes so as to become PCV2-positive. The offspring was investigated for the development of PMWS. A strong protective effect of vaccination was observed regarding reproductive disorders in sows since the number of mummified and stillborn piglets was similar for vaccinated PCV2-infected sows and PCV2-negative control sows while it was higher for PCV2-infected non-vaccinated sows. No significant difference was noted between the two protocol of vaccination except for PCV2 titers in piglets which were a bit lower with the bivalent vaccine. Cross-fostering of piglets born of infected sows and deprived of colostrum resulted in the rapid horizontal transmission of PCV2. Among the 84 piglets born during the study, 16 of them developed PMWS. Among these PMWS-affected piglets, most of them were born of PCV2-infected non-vaccinated sows and had no passive immunity. Improper vaccination for parvovirus/erisypelas and cross-fostering piglets between sows with undetermined immune status for PCV2 are two risk factors for PMWS.
